Receptor - Tyrosine Phosphatase Lar in Lung Development

Yu, Qiang

Abstract

Funding Agency

• Agency: National Institute of Health (NIH)
• Institute: National Heart, Lung, and Blood Institute (NHLBI)
• Type: Research Project (R01)
• Project #: 3R01HL054044-01A1S1
• Application #: 1073100
• Study Section: Lung Biology and Pathology Study Section (LBPA)
• Project Start: 1996-01-01
• Project End: 1999-12-31
• Budget Start: 1996-01-01
• Budget End: 1996-12-31
• Support Year: 1
• Fiscal Year: 1996

Institution

• Name: Boston University
• Department: Internal Medicine/Medicine
• Type: Schools of Medicine
• DUNS #: 604483045
• City: Boston
• State: MA
• Country: United States
• Zip Code: 02118

Publications

Wang, X; Weng, L P; Yu, Q (2000) Specific inhibition of FGF-induced MAPK activation by the receptor-like protein tyrosine phosphatase LAR. Oncogene 19:2346-53

Matsui, R; Brody, J S; Yu, Q (1999) FGF-2 induces surfactant protein gene expression in foetal rat lung epithelial cells through a MAPK-independent pathway. Cell Signal 11:221-8

Weng, L P; Wang, X; Yu, Q (1999) Transmembrane tyrosine phosphatase LAR induces apoptosis by dephosphorylating and destabilizing p130Cas. Genes Cells 4:185-96

Weng, L P; Yuan, J; Yu, Q (1998) Overexpression of the transmembrane tyrosine phosphatase LAR activates the caspase pathway and induces apoptosis. Curr Biol 8:247-56