Agency
National Institute of Health (NIH)
Institute
National Institute of Neurological Disorders and Stroke (NINDS)
Type
Research Project (R01)
Project #
5R01NS017117-13
Application #
2263147
Study Section
Neurological Sciences Subcommittee 1 (NLS)
Project Start
1981-04-01
Project End
1995-12-31
Budget Start
1994-07-01
Budget End
1995-12-31
Support Year
13
Fiscal Year
1994
Total Cost
Indirect Cost
Name
Children's Hospital of Los Angeles
Department
Type
DUNS #
094878337
City
Los Angeles
State
CA
Country
United States
Zip Code
90027
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Banerjee, P K; Olsen, R W; Tillakaratne, N J et al. (1998) Absence seizures decrease steroid modulation of t-[35S]butylbicyclophosphorothionate binding in thalamic relay nuclei. J Pharmacol Exp Ther 287:766-72
Banerjee, P K; Tillakaratne, N J; Brailowsky, S et al. (1998) Alterations in GABAA receptor alpha 1 and alpha 4 subunit mRNA levels in thalamic relay nuclei following absence-like seizures in rats. Exp Neurol 154:213-23
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Snead 3rd, O C (1996) Relation of the [3H] gamma-hydroxybutyric acid (GHB) binding site to the gamma-aminobutyric acidB (GABAB) receptor in rat brain. Biochem Pharmacol 52:1235-43
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Banerjee, P K; Snead 3rd, O C (1995) Presynaptic gamma-hydroxybutyric acid (GHB) and gamma-aminobutyric acidB (GABAB) receptor-mediated release of GABA and glutamate (GLU) in rat thalamic ventrobasal nucleus (VB): a possible mechanism for the generation of absence-like seizures induced by GH J Pharmacol Exp Ther 273:1534-43
Snead 3rd, O C (1995) Basic mechanisms of generalized absence seizures. Ann Neurol 37:146-57
Banerjee, P K; Snead 3rd, O C (1995) Thalamic NMDA receptors in the gamma-hydroxybutyrate model of absence seizures: a cerebral microinjection study in rats. Neuropharmacology 34:43-53
Banerjee, P K; Snead 3rd, O C (1994) Thalamic mediodorsal and intralaminar nuclear lesions disrupt the generation of experimentally induced generalized absence-like seizures in rats. Epilepsy Res 17:193-205

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