Neuropeptides and Spinal Mechanisms of Hyperalgesia - Richard Traub

Abstract

Funding Agency

Agency: National Institute of Health (NIH)
Institute: National Institute of Neurological Disorders and Stroke (NINDS)
Type: Research Project (R01)
Project #: 3R01NS030604-04S1
Application #: 2631887
Study Section: Neurological Sciences Subcommittee 1 (NLS)

Project Start: 1993-07-01
Project End: 1998-06-30
Budget Start: 1996-07-01
Budget End: 1998-06-30
Support Year: 4
Fiscal Year: 1997
Total Cost
Indirect Cost

Institution

Name: University of Maryland Baltimore
Department: Pharmacology
Type: Schools of Dentistry
DUNS #: 003255213

City: Baltimore
State: MD
Country: United States
Zip Code: 21201

Related projects


NIH 1997 R01 NS	Neuropeptides and Spinal Mechanisms of Hyperalgesia Traub, Richard J. / University of Iowa
NIH 1997 R01 NS	Neuropeptides and Spinal Mechanisms of Hyperalgesia Traub, Richard J. / University of Maryland Baltimore
NIH 1996 R01 NS	Neuropeptides and Spinal Mechanisms of Hyperalgesia Traub, Richard J. / University of Maryland Baltimore
NIH 1995 R01 NS	Neuropeptides and Spinal Mechanisms of Hyperalgesia Traub, Richard J. / University of Iowa
NIH 1994 R01 NS	Neuropeptides and Spinal Mechanisms of Hyperalgesia Traub, Richard J. / University of Iowa
NIH 1993 R01 NS	Neuropeptides and Spinal Mechanisms of Hyperalgesia Traub, Richard J. / University of Iowa

Publications

Traub, R J; Solodkin, A; Meller, S T et al. (1994) Spinal cord NADPH-diaphorase histochemical staining but not nitric oxide synthase immunoreactivity increases following carrageenan-produced hindpaw inflammation in the rat. Brain Res 668:204-10

Traub, R J; Lim, F; Sengupta, J N et al. (1994) Noxious distention of viscera results in differential c-Fos expression in second order sensory neurons receiving 'sympathetic' or 'parasympathetic' input. Neurosci Lett 180:71-5

Traub, R J; Solodkin, A; Gebhart, G F (1994) NADPH-diaphorase histochemistry provides evidence for a bilateral, somatotopically inappropriate response to unilateral hindpaw inflammation in the rat. Brain Res 647:113-23

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