Mechanisms of Ischemic Brain Damage

Quast, Michael J.

Abstract

Funding Agency

• Agency: National Institute of Health (NIH)
• Institute: National Institute of Neurological Disorders and Stroke (NINDS)
• Type: Research Project (R01)
• Project #: 5R01NS033575-02
• Application #: 2272442
• Study Section: Neurology B Subcommittee 2 (NEUB)
• Project Start: 1995-06-15
• Project End: 1998-05-31
• Budget Start: 1996-06-01
• Budget End: 1997-05-31
• Support Year: 2
• Fiscal Year: 1996

Institution

• Name: University of Texas Medical Br Galveston
• Department: Zoology
• Type: Schools of Medicine
• DUNS #: 041367053
• City: Galveston
• State: TX
• Country: United States
• Zip Code: 77555

Publications

Wei, J; Quast, M J (1998) Effect of nitric oxide synthase inhibitor on a hyperglycemic rat model of reversible focal ischemia: detection of excitatory amino acids release and hydroxyl radical formation. Brain Res 791:146-56

Wei, J; Huang, N C; Quast, M J (1997) Hydroxyl radical formation in hyperglycemic rats during middle cerebral artery occlusion/reperfusion. Free Radic Biol Med 23:986-95

Mottet, I; Quast, M J; Dewitt, D S et al. (1997) NG-nitro-L-arginine methyl ester modifies the input function measured by dynamic susceptibility contrast magnetic resonance imaging. J Cereb Blood Flow Metab 17:791-800

Quast, M J; Wei, J; Huang, N C et al. (1997) Perfusion deficit parallels exacerbation of cerebral ischemia/reperfusion injury in hyperglycemic rats. J Cereb Blood Flow Metab 17:553-9

Huang, N C; Wei, J; Quast, M J (1996) A comparison of the early development of ischemic brain damage in normoglycemic and hyperglycemic rats using magnetic resonance imaging. Exp Brain Res 109:33-42