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Stewart, Richard S; Harris, David A (2005) A transmembrane form of the prion protein is localized in the Golgi apparatus of neurons. J Biol Chem 280:15855-64
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Stewart, Richard S; Harris, David A (2003) Mutational analysis of topological determinants in prion protein (PrP) and measurement of transmembrane and cytosolic PrP during prion infection. J Biol Chem 278:45960-8
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Drisaldi, Bettina; Stewart, Richard S; Adles, Cheryl et al. (2003) Mutant PrP is delayed in its exit from the endoplasmic reticulum, but neither wild-type nor mutant PrP undergoes retrotranslocation prior to proteasomal degradation. J Biol Chem 278:21732-43
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Stewart, R S; Harris, D A (2001) Most pathogenic mutations do not alter the membrane topology of the prion protein. J Biol Chem 276:2212-20
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Ivanova, L; Barmada, S; Kummer, T et al. (2001) Mutant prion proteins are partially retained in the endoplasmic reticulum. J Biol Chem 276:42409-21
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Stewart, R S; Drisaldi, B; Harris, D A (2001) A transmembrane form of the prion protein contains an uncleaved signal peptide and is retained in the endoplasmic Reticulum. Mol Biol Cell 12:881-9
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Soto, C; Kascsak, R J; Saborio, G P et al. (2000) Reversion of prion protein conformational changes by synthetic beta-sheet breaker peptides. Lancet 355:192-7
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Chiesa, R; Harris, D A (2000) Nerve growth factor-induced differentiation does not alter the biochemical properties of a mutant prion protein expressed in PC12 cells. J Neurochem 75:72-80
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Narwa, R; Harris, D A (1999) Prion proteins carrying pathogenic mutations are resistant to phospholipase cleavage of their glycolipid anchors. Biochemistry 38:8770-7
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Daude, N; Lehmann, S; Harris, D A (1997) Identification of intermediate steps in the conversion of a mutant prion protein to a scrapie-like form in cultured cells. J Biol Chem 272:11604-12
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Showing the most recent 10 out of 12 publications
