Agency
National Institute of Health (NIH)
Institute
National Institute of Neurological Disorders and Stroke (NINDS)
Type
Research Project (R01)
Project #
1R01NS035607-01
Application #
2274856
Study Section
Special Emphasis Panel (ZRG1-NLS-3 (01))
Project Start
1996-09-01
Project End
1999-07-31
Budget Start
1996-09-01
Budget End
1997-07-31
Support Year
1
Fiscal Year
1996
Total Cost
Indirect Cost
Name
University of Kentucky
Department
Physiology
Type
Schools of Medicine
DUNS #
832127323
City
Lexington
State
KY
Country
United States
Zip Code
40506
Bruckner, S R; Tammariello, S P; Kuan, C Y et al. (2001) JNK3 contributes to c-Jun activation and apoptosis but not oxidative stress in nerve growth factor-deprived sympathetic neurons. J Neurochem 78:298-303
Tucker, H M; Kihiko, M; Caldwell, J N et al. (2000) The plasmin system is induced by and degrades amyloid-beta aggregates. J Neurosci 20:3937-46
Kihiko, M E; Tucker, H M; Rydel, R E et al. (1999) c-Jun contributes to amyloid beta-induced neuronal apoptosis but is not necessary for amyloid beta-induced c-jun induction. J Neurochem 73:2609-12
Tucker, H M; Rydel, R E; Wright, S et al. (1998) Human amylin induces ""apoptotic"" pattern of gene expression concomitant with cortical neuronal apoptosis. J Neurochem 71:506-16