The University of Chicago Post-Baccalaureate Research Education Program (PREP) is designed to provide research training and enrichment activities to recent college graduates from groups underrepresented in the biomedical and behavioral sciences so they will be better prepared to apply, matriculate, and succeed in a PhD program. It is based on the concept that an in-depth mentored research experience, along with enrichment activities that help develop identity as a scientist, is the optimal preparation for graduate studies in the biomedical sciences. The background and research interests of each participant are considered when selecting Mentors for one year of laboratory-based research, with Mentors who have outstanding records of promoting the training, education, and career advancement of biomedical scientists. The research experience is complemented by a multi-tiered skill-enhancement program for improving oral and written communication, critical thinking and analysis, and ethical decision-making. Training in such skills is provided by a year-long PREP Journal Club, numerous workshops, a writing course, an ethics course, academic courses, and other skill-building activities. Although the Program is highly structured and logically organized, it is flexible in that an Individual Development Plan (IDP) serves as the roadmap for each Scholar's course over their tenure in the University of Chicago PREP. The Program and its components are assessed at specific intervals throughout the year, including both internal and external evaluations. The ultimate success will be measured by the matriculation of Scholars into PhD programs, their completion of PhD programs, and their long-term successes as biomedical scientists.
The goal of the University of Chicago Post-Baccalaureate Research Education Program (PREP) is to help diversify the scientific workforce. The PREP provides mentored research experience in a biomedical laboratory and academic enhancements in the form of skill-building workshops and courses, in order to prepare postbac students in the biomedical and behavioral sciences for admission to PhD programs at competitive research institutions. To assess the success of this endeavor, the participants' progress will be followed throughout their graduate programs and subsequent career path.
|Domowicz, Miriam; Wadlington, Natasha L; Henry, Judith G et al. (2018) Glial cell responses in a murine multifactorial perinatal brain injury model. Brain Res 1681:52-63|
|Bhanvadia, Raj R; VanOpstall, Calvin; Brechka, Hannah et al. (2018) MEIS1 and MEIS2 Expression and Prostate Cancer Progression: A Role For HOXB13 Binding Partners in Metastatic Disease. Clin Cancer Res 24:3668-3680|
|Umana, Iboro C; Daniele, Claire A; Miller, Brooke A et al. (2017) Nicotinic modulation of descending pain control circuitry. Pain 158:1938-1950|
|Mir-Sanchis, Ignacio; Roman, Christina A; Misiura, Agnieszka et al. (2016) Staphylococcal SCCmec elements encode an active MCM-like helicase and thus may be replicative. Nat Struct Mol Biol 23:891-898|
|Lamar, Kay-Marie; Bogdanovich, Sasha; Gardner, Brandon B et al. (2016) Overexpression of Latent TGF? Binding Protein 4 in Muscle Ameliorates Muscular Dystrophy through Myostatin and TGF?. PLoS Genet 12:e1006019|
|Riley, Sean P; Patterson, Jennifer L; Nava, Samantha et al. (2014) Pathogenic Rickettsia species acquire vitronectin from human serum to promote resistance to complement-mediated killing. Cell Microbiol 16:849-61|
|Goldstein, Jeffery A; Bogdanovich, Sasha; Beiriger, Anastasia et al. (2014) Excess SMAD signaling contributes to heart and muscle dysfunction in muscular dystrophy. Hum Mol Genet 23:6722-31|
|Sittig, Laura J; Jeong, Choongwon; Tixier, Emily et al. (2014) Phenotypic instability between the near isogenic substrains BALB/cJ and BALB/cByJ. Mamm Genome 25:564-72|
|Skinner, John J; Yu, Wookyung; Gichana, Elizabeth K et al. (2014) Benchmarking all-atom simulations using hydrogen exchange. Proc Natl Acad Sci U S A 111:15975-80|
|Makinen, Marvin W; Bamba, Ravinder; Ikejimba, Lynda et al. (2013) The vanadyl chelate bis(acetylacetonato)oxovanadium(IV) increases the fractional uptake of 2-(fluorine-18)-2-deoxy-D-glucose by cultured human breast carcinoma cells. Dalton Trans 42:11862-7|
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