Agency
National Institute of Health (NIH)
Institute
National Cancer Institute (NCI)
Type
First Independent Research Support & Transition (FIRST) Awards (R29)
Project #
5R29CA078214-04
Application #
6173694
Study Section
Molecular Biology Study Section (MBY)
Program Officer
Mietz, Judy
Project Start
1997-09-30
Project End
2002-07-31
Budget Start
2000-08-01
Budget End
2001-07-31
Support Year
4
Fiscal Year
2000
Total Cost
$105,706
Indirect Cost
Name
University of South Florida
Department
Biochemistry
Type
Schools of Medicine
DUNS #
City
Tampa
State
FL
Country
United States
Zip Code
33612
Savell, Jason; Ma, Yihong; Morrow, Kristin S et al. (2004) AG490 inhibits G1-S traverse in BALB/c-3T3 cells following either mitogenic stimulation or exogenous expression of E2F-1. Mol Cancer Ther 3:205-13
Ma, Yihong; Cress, W Douglas; Haura, Eric B (2003) Flavopiridol-induced apoptosis is mediated through up-regulation of E2F1 and repression of Mcl-1. Mol Cancer Ther 2:73-81
Ma, Yihong; Yuan, Jing; Huang, Mei et al. (2003) Regulation of the cyclin D3 promoter by E2F1. J Biol Chem 278:16770-6
Croxton, Rhonda; Ma, Yihong; Song, Lanxi et al. (2002) Direct repression of the Mcl-1 promoter by E2F1. Oncogene 21:1359-69
Ma, Yihong; Croxton, Rhonda; Moorer Jr, Ronnie L et al. (2002) Identification of novel E2F1-regulated genes by microarray. Arch Biochem Biophys 399:212-24
Croxton, Rhonda; Ma, Yihong; Cress, W Douglas (2002) Differences in DNA binding properties between E2F1 and E2F4 specify repression of the Mcl-1 promoter. Oncogene 21:1563-70
He, Yiwen; Cress, W Douglas (2002) E2F-3B is a physiological target of cyclin A. J Biol Chem 277:23493-9
He, Y; Armanious, M K; Thomas, M J et al. (2000) Identification of E2F-3B, an alternative form of E2F-3 lacking a conserved N-terminal region. Oncogene 19:3422-33