Murine Host Response to Porphyromonas Gingivalis

Baker, Pamela J.

Abstract

Funding Agency

• Agency: National Institute of Health (NIH)
• Institute: National Institute of Dental & Craniofacial Research (NIDCR)
• Type: First Independent Research Support & Transition (FIRST) Awards (R29)
• Project #: 5R29DE010728-03
• Application #: 2131630
• Study Section: Oral Biology and Medicine Subcommittee 1 (OBM)
• Project Start: 1994-05-01
• Project End: 1999-04-30
• Budget Start: 1996-05-01
• Budget End: 1997-04-30
• Support Year: 3
• Fiscal Year: 1996

Institution

• Name: Bates College
• Department: Biology
• Type: Schools of Arts and Sciences
• DUNS #: 058951401
• City: Lewiston
• State: ME
• Country: United States
• Zip Code: 04240

Publications

Baker, P J; DuFour, L; Dixon, M et al. (2000) Adhesion molecule deficiencies increase Porphyromonas gingivalis-induced alveolar bone loss in mice. Infect Immun 68:3103-7

Baker, P J; Dixon, M; Evans, R T et al. (2000) Heterogeneity of Porphyromonas gingivalis strains in the induction of alveolar bone loss in mice. Oral Microbiol Immunol 15:27-32

Baker, P J; Dixon, M; Roopenian, D C (2000) Genetic control of susceptibility to Porphyromonas gingivalis-induced alveolar bone loss in mice. Infect Immun 68:5864-8

Baker, P J (2000) The role of immune responses in bone loss during periodontal disease. Microbes Infect 2:1181-92

Baker, P J; Dixon, M; Evans, R T et al. (1999) CD4(+) T cells and the proinflammatory cytokines gamma interferon and interleukin-6 contribute to alveolar bone loss in mice. Infect Immun 67:2804-9

Baker, P J; Carter, S; Dixon, M et al. (1999) Serum antibody response to oral infection precedes but does not prevent Porphyromonas gingivalis-induced alveolar bone loss in mice. Oral Microbiol Immunol 14:194-6