Agency
National Institute of Health (NIH)
Institute
National Institute of Diabetes and Digestive and Kidney Diseases (NIDDK)
Type
First Independent Research Support & Transition (FIRST) Awards (R29)
Project #
1R29DK046459-01A3
Application #
2145683
Study Section
Special Emphasis Panel (ZRG4-GMA-1 (01))
Project Start
1996-03-15
Project End
2001-02-28
Budget Start
1996-03-15
Budget End
1997-02-28
Support Year
1
Fiscal Year
1996
Total Cost
Indirect Cost
Name
Cedars-Sinai Medical Center
Department
Type
DUNS #
075307785
City
Los Angeles
State
CA
Country
United States
Zip Code
90048
Lin, Henry C; Taylor, Ian L (2004) Release of peptide YY by fat in the proximal but not distal gut depends on an atropine-sensitive cholinergic pathway. Regul Pept 117:73-6
Lin, H C; Chen, J H (2003) Slowing of intestinal transit by fat depends on an ondansetron - sensitive, efferent serotonergic pathway. Neurogastroenterol Motil 15:317-22
Lin, Henry C; Neevel, Corynn; Chen, Peng-Sheng et al. (2003) Slowing of intestinal transit by fat or peptide YY depends on beta-adrenergic pathway. Am J Physiol Gastrointest Liver Physiol 285:G1310-6
Lin, Henry C; Chey, William Y (2003) Cholecystokinin and peptide YY are released by fat in either proximal or distal small intestine in dogs. Regul Pept 114:131-5
Lin, H C; Perdomo, O L; Zhao, X T (2001) Intestinal transit in dogs is accelerated by volume distension during fat-induced jejunal brake. Dig Dis Sci 46:19-23
Furness, J B; Koopmans, H S; Robbins, H L et al. (2000) Identification of intestinofugal neurons projecting to the coeliac and superior mesenteric ganglia in the rat. Auton Neurosci 83:81-5
Zhao, X T; Wang, L; Lin, H C (2000) Slowing of intestinal transit by fat depends on naloxone-blockable efferent, opioid pathway. Am J Physiol Gastrointest Liver Physiol 278:G866-70
Zhao, X T; Walsh, J H; Wong, H et al. (1999) Intestinal fat-induced inhibition of meal-stimulated gastric acid secretion depends on CCK but not peptide YY. Am J Physiol 276:G550-5
Lin, H C; Zhao, X T; Chu, A W et al. (1997) Fiber-supplemented enteral formula slows intestinal transit by intensifying inhibitory feedback from the distal gut. Am J Clin Nutr 65:1840-4
Lin, H C; Zhao, X T; Wang, L (1997) Intestinal transit is more potently inhibited by fat in the distal (ileal brake) than in the proximal (jejunal brake) gut. Dig Dis Sci 42:19-25