Riboflavin and Flavoprotein Gene Expression

Ross, Nathan S.

Abstract

Funding Agency

• Agency: National Institute of Health (NIH)
• Institute: Eunice Kennedy Shriver National Institute of Child Health & Human Development (NICHD)
• Type: First Independent Research Support & Transition (FIRST) Awards (R29)
• Project #: 5R29HD025299-02
• Application #: 3470061
• Study Section: Special Emphasis Panel (SRC (06))
• Project Start: 1988-12-01
• Project End: 1990-08-31
• Budget Start: 1989-12-01
• Budget End: 1990-08-31
• Support Year: 2
• Fiscal Year: 1990

Institution

• Name: Case Western Reserve University
• Type: Schools of Medicine
• DUNS #: 077758407
• City: Cleveland
• State: OH
• Country: United States
• Zip Code: 44106

Publications

Ross, N S (1994) Epidemiology of Cushing's syndrome and subclinical disease. Endocrinol Metab Clin North Am 23:539-46

Geffner, M E; Su, F; Ross, N S et al. (1993) An arginine to histidine mutation in codon 311 of the C-erbA beta gene results in a mutant thyroid hormone receptor that does not mediate a dominant negative phenotype. J Clin Invest 91:538-46

Pang, X P; Ross, N S; Park, M et al. (1992) Tumor necrosis factor-alpha activates nuclear factor kappa B and induces manganous superoxide dismutase and phosphodiesterase mRNA in human papillary thyroid carcinoma cells. J Biol Chem 267:12826-30

Ross, N S; Hansen, T P (1992) Riboflavin deficiency is associated with selective preservation of critical flavoenzyme-dependent metabolic pathways. Biofactors 3:185-90

Ross, N S; Klein, M R (1990) Riboflavin deficiency in cultured rat hepatoma cells: a model for studying the hepatic effects of riboflavin deficiency. In Vitro Cell Dev Biol 26:280-4

Ross, N S; Aron, D C (1990) Hormonal evaluation of the patient with an incidentally discovered adrenal mass. N Engl J Med 323:1401-5