Funding Agency

Agency
National Institute of Health (NIH)
Institute
National Heart, Lung, and Blood Institute (NHLBI)
Type
First Independent Research Support & Transition (FIRST) Awards (R29)
Project #
1R29HL051129-01A2
Application #
2227664
Study Section
Cardiovascular and Renal Study Section (CVB)
Project Start
1996-03-01
Project End
2001-02-28
Budget Start
1996-03-01
Budget End
1997-02-28
Support Year
1
Fiscal Year
1996
Total Cost
Indirect Cost

  Institution

Name
University of California Los Angeles
Department
Internal Medicine/Medicine
Type
Schools of Medicine
DUNS #
119132785
City
Los Angeles
State
CA
Country
United States
Zip Code
90095

  Publications

Goldhaber, Joshua I; Xie, Lai-Hua; Duong, Tan et al. (2005) Action potential duration restitution and alternans in rabbit ventricular myocytes: the key role of intracellular calcium cycling. Circ Res 96:459-66
Fukumoto, Gary H; Lamp, Scott T; Motter, Christi et al. (2005) Metabolic inhibition alters subcellular calcium release patterns in rat ventricular myocytes: implications for defective excitation-contraction coupling during cardiac ischemia and failure. Circ Res 96:551-7
Reuter, Hannes; Han, Tieyan; Motter, Christi et al. (2004) Mice overexpressing the cardiac sodium-calcium exchanger: defects in excitation-contraction coupling. J Physiol 554:779-89
Korge, P; Goldhaber, J I; Weiss, J N (2001) Phenylarsine oxide induces mitochondrial permeability transition, hypercontracture, and cardiac cell death. Am J Physiol Heart Circ Physiol 280:H2203-13
Goldhaber, J I; Qayyum, M S (2000) Oxygen free radicals and excitation-contraction coupling. Antioxid Redox Signal 2:55-64
Kondo, R P; Weiss, J N; Goldhaber, J I (2000) Putative ryanodine receptors in the sarcolemma of ventricular myocytes. Pflugers Arch 440:125-31
Goldhaber, J I; Lamp, S T; Walter, D O et al. (1999) Local regulation of the threshold for calcium sparks in rat ventricular myocytes: role of sodium-calcium exchange. J Physiol 520 Pt 2:431-8
Honda, H M; Wakamatsu, B K; Goldhaber, J I et al. (1999) High-density lipoprotein increases intracellular calcium levels by releasing calcium from internal stores in human endothelial cells. Atherosclerosis 143:299-306