Regulation and Expression of Biological Processes - G. Hatfield

Abstract

Funding Agency

Agency: National Institute of Health (NIH)
Institute: National Institute of General Medical Sciences (NIGMS)
Type: Institutional National Research Service Award (T32)
Project #: 2T32GM007134-16
Application #: 3536841
Study Section: Genetic Basis of Disease Review Committee (GBD)

Project Start: 1975-07-01
Project End: 1995-06-30
Budget Start: 1990-07-01
Budget End: 1991-06-30
Support Year: 16
Fiscal Year: 1990
Total Cost
Indirect Cost

Institution

Name: University of California Irvine
Department
Type: Schools of Medicine
DUNS #: 161202122

City: Irvine
State: CA
Country: United States
Zip Code: 92697

Related projects


NIH 1994 T32 GM	Regulation & Expression of Biological Processes Hatfield, G. Wesley / University of California Irvine
NIH 1993 T32 GM	Regulation & Expression of Biological Processes Hatfield, G. Wesley / University of California Irvine
NIH 1992 T32 GM	Regulation and Expression of Biological Processes Hatfield, G. Wesley / University of California Irvine
NIH 1991 T32 GM	Regulation and Expression of Biological Processes Hatfield, G. Wesley / University of California Irvine
NIH 1990 T32 GM	Regulation and Expression of Biological Processes Hatfield, G. Wesley / University of California Irvine
NIH 1989 T32 GM	Regulation of Biological Processes Hatfield, G. Wesley / University of California Irvine
NIH 1988 T32 GM	Regulation of Biological Processes Hatfield, G. Wesley / University of California Irvine
NIH 1987 T32 GM	Regulation of Biological Processes Sypherd, Paul S. / University of California Irvine
NIH 1986 T32 GM	Regulation of Biological Processes Sypherd, Paul S. / University of California Irvine
NIH 1985 T32 GM	Regulation of Biological Processes Sypherd, Paul S. / University of California Irvine

Publications

Granger, S W; Fan, H (2001) Purification of Moloney murine leukemia virus chromatin from infected cells by an affinity method. J Biomed Sci 8:278-89

Lander, J K; Chesebro, B; Fan, H (1999) Appearance of mink cell focus-inducing recombinants during in vivo infection by moloney murine leukemia virus (M-MuLV) or the Mo+PyF101 M-MuLV enhancer variant: implications for sites of generation and roles in leukemogenesis. J Virol 73:5671-80

Lander, J K; Fan, H (1997) Low-frequency loss of heterozygosity in Moloney murine leukemia virus-induced tumors in BRAKF1/J mice. J Virol 71:3940-52

Piedras-Renteria, E S; Chen, C C; Best, P M (1997) Antisense oligonucleotides against rat brain alpha1E DNA and its atrial homologue decrease T-type calcium current in atrial myocytes. Proc Natl Acad Sci U S A 94:14936-41

Feuer, G; Lee, W T; Pattengale, P K et al. (1993) Specificity of disease induced by M-MuLV chimeric retroviruses containing v-myc or v-src is not determined by the LTR. Virology 195:286-91

Feuer, G; Fan, H (1990) Substitution of murine transthyretin (prealbumin) regulatory sequences into the Moloney murine leukemia virus long terminal repeat yields infectious virus with altered biological properties. J Virol 64:6130-40

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