This is a new training program in Neonatal and Developmental Diseases which will be established within UCLA School of Medicine's existing Neonatology Program. This is a five year program geared towards training postresident neonatology fellows (M.D. and M.D., Ph.D.) with the idea of developing independent and productive physician- scientists and training of postdoctoral Ph.D. fellows in the area of neonatal and developmental diseases. This program will strive to attract individuals with a commitment towards academic medicine, provide a scientifically rich environment where M.D. and Ph.D. postresident and postdoctoral trainees will be afforded the opportunity of training side-by-side in established laboratories with expertise in developmental biology, genetics, molecular biology, and cell biology. Program faculty have been chosen from diverse fields and from various departments at the UCLA School of Medicine, with a Nationally reputable research program and a proven track record in training scientists and clinicians. The program is structured to include clinical neonatology training (as per the requirements spelled out by the American Board of Pediatrics) a bulk of which will occur during the first year, the second and third years will be devoted to training in laboratory-based research for the postresidency fellows. Following this initial training period, and after rigorous evaluation, the postresidency fellows will be allowed to progress into the postdoctoral portion of their fellowship that will consist of two additional years (four and five) following which they will be expected to obtain independent funding for their research programs. This training program requests support from NICHD for only years two, three and four. In addition, postdoctoral Ph.D. fellows will be afforded a three year training period, of which this program requests funds for only the first year, with the expectation that they will obtain independent postdoctoral fellowship support from independent applications for support. Given the available faculty mentors, the facilities, the structure, a formal evaluation process, and the expectations both at recruitment and throughout the training period, this training program is expected to produce independent investigators in the field of Neonatal and Developmental Diseases.

Agency
National Institute of Health (NIH)
Institute
Eunice Kennedy Shriver National Institute of Child Health & Human Development (NICHD)
Type
Institutional National Research Service Award (T32)
Project #
5T32HD007549-03
Application #
6625193
Study Section
Pediatrics Subcommittee (CHHD)
Program Officer
Raju, Tonse N
Project Start
2001-07-01
Project End
2006-04-30
Budget Start
2003-05-01
Budget End
2004-04-30
Support Year
3
Fiscal Year
2003
Total Cost
$358,011
Indirect Cost
Name
University of California Los Angeles
Department
Pediatrics
Type
Schools of Medicine
DUNS #
092530369
City
Los Angeles
State
CA
Country
United States
Zip Code
90095
Calkins, Kara L; Sanchez, Lauren A; Tseng, Chi-Hong et al. (2016) Effect of High-Dose Cysteine Supplementation on Erythrocyte Glutathione: A Double-Blinded, Randomized Placebo-Controlled Pilot Study in Critically Ill Neonates. JPEN J Parenter Enteral Nutr 40:226-34
Gibson, Leena Caroline; Shin, Bo-Chul; Dai, Yun et al. (2015) Early leptin intervention reverses perturbed energy balance regulating hypothalamic neuropeptides in the pre- and postnatal calorie-restricted female rat offspring. J Neurosci Res 93:902-12
Farzin, Azadeh; Boyer, Pamela; Ank, Bonnie et al. (2013) Amniotic fluid exhibits an innate inhibitory activity against HIV type 1 replication in vitro. AIDS Res Hum Retroviruses 29:77-83
Abbasi, Afshan; Thamotharan, Manikkavasagar; Shin, Bo-Chul et al. (2012) Myocardial macronutrient transporter adaptations in the adult pregestational female intrauterine and postnatal growth-restricted offspring. Am J Physiol Endocrinol Metab 302:E1352-62
Fung, Camille; Evans, Edward; Shin, Don et al. (2010) Hypoxic-ischemic brain injury exacerbates neuronal apoptosis and precipitates spontaneous seizures in glucose transporter isoform 3 heterozygous null mice. J Neurosci Res 88:3386-98
Zhao, Y; Fung, C; Shin, D et al. (2010) Neuronal glucose transporter isoform 3 deficient mice demonstrate features of autism spectrum disorders. Mol Psychiatry 15:286-99
Soliman, Antoine; Michelsen, Kathrin S; Karahashi, Hisae et al. (2010) Platelet-activating factor induces TLR4 expression in intestinal epithelial cells: implication for the pathogenesis of necrotizing enterocolitis. PLoS One 5:e15044
Evans, E; Koh, S; Lerner, Jt et al. (2010) Accuracy of amplitude integrated EEG in a neonatal cohort. Arch Dis Child Fetal Neonatal Ed 95:F169-73
Kurlawalla-Martinez, Christine; Stiles, Bangyan; Wang, Ying et al. (2005) Insulin hypersensitivity and resistance to streptozotocin-induced diabetes in mice lacking PTEN in adipose tissue. Mol Cell Biol 25:2498-510
Widney, Daniel P; Hu, Yan; Foreman-Wykert, Amy K et al. (2005) CXCR3 and its ligands participate in the host response to Bordetella bronchiseptica infection of the mouse respiratory tract but are not required for clearance of bacteria from the lung. Infect Immun 73:485-93

Showing the most recent 10 out of 16 publications