Abstract

The function of this Administration Core is to provide the UCACE Director with the organizational, administrative, and secretarial support to enable the scientific and clinical research goals of the program to be accomplished. The UCACE Office will be located within the ample office space allocated to the Section of Rheumatology, adjacent to the laboratories of Drs. Clark, Wilson and Weigert. Specific responsibilities of this core, embodied within the UCACE Office are as follows: 1. Coordinate the scientific activities of the program 2. Coordinate the clinical trials program 3. Oversee the documentation related to the clinical trials program 4. Facilitate interactions between investigators at the formal and informal level 5. Coordinate a standing External Scientific Advisory Committee. 6. Prepare annual progress reports 7. Organize annual formal review 8. Organize visits of other external scientists and consultants 9. Provide day to day administrative (fiscal and secretarial) support to investigators 10. Organize and administer monthly UCACE Research Seminars 11. Keep accurate records and accounts on resource utilization 12. Arrange for yearly travel of Director and Project/Core Leaders to national meetings Support services will be provided equally to all Projects and Cores.

  Funding Agency

Agency
National Institute of Health (NIH)
Institute
National Institute of Allergy and Infectious Diseases (NIAID)
Type
Research Program--Cooperative Agreements (U19)
Project #
1U19AI082724-01
Application #
7688952
Study Section
Special Emphasis Panel (ZAI1-QV-I (J3))
Project Start
2009-06-10
Project End
2014-05-31
Budget Start
2009-06-10
Budget End
2010-05-31
Support Year
1
Fiscal Year
2009
Total Cost
$107,193
Indirect Cost

  Institution

Name
University of Chicago
Department
Type
DUNS #
005421136
City
Chicago
State
IL
Country
United States
Zip Code
60637

  Publications

Kinloch, Andrew J; Kaiser, Ylva; Wolfgeher, Don et al. (2018) In Situ Humoral Immunity to Vimentin in HLA-DRB1*03+ Patients With Pulmonary Sarcoidosis. Front Immunol 9:1516
Chen, Yao-Qing; Wohlbold, Teddy John; Zheng, Nai-Ying et al. (2018) Influenza Infection in Humans Induces Broadly Cross-Reactive and Protective Neuraminidase-Reactive Antibodies. Cell 173:417-429.e10
Henry, Carole; Palm, Anna-Karin E; Krammer, Florian et al. (2018) From Original Antigenic Sin to the Universal Influenza Virus Vaccine. Trends Immunol 39:70-79
Wilson, Patrick C; Cobey, Sarah (2018) Characterization of the immunologic repertoire: A quick start guide. Immunol Rev 284:5-8
Stamper, Christopher T; Wilson, Patrick C (2018) What Are the Primary Limitations in B-Cell Affinity Maturation, and How Much Affinity Maturation Can We Drive with Vaccination? Is Affinity Maturation a Self-Defeating Process for Eliciting Broad Protection? Cold Spring Harb Perspect Biol 10:
Leon, Paul E; Wohlbold, Teddy John; He, Wenqian et al. (2017) Generation of Escape Variants of Neutralizing Influenza Virus Monoclonal Antibodies. J Vis Exp :
He, Wenqian; Chen, Chi-Jene; Mullarkey, Caitlin E et al. (2017) Alveolar macrophages are critical for broadly-reactive antibody-mediated protection against influenza A virus in mice. Nat Commun 8:846
DiPiazza, Anthony; Nogales, Aitor; Poulton, Nicholas et al. (2017) Pandemic 2009 H1N1 Influenza Venus reporter virus reveals broad diversity of MHC class II-positive antigen-bearing cells following infection in vivo. Sci Rep 7:10857
Lau, Denise; Lan, Linda Yu-Ling; Andrews, Sarah F et al. (2017) Low CD21 expression defines a population of recent germinal center graduates primed for plasma cell differentiation. Sci Immunol 2:
Bunker, Jeffrey J; Erickson, Steven A; Flynn, Theodore M et al. (2017) Natural polyreactive IgA antibodies coat the intestinal microbiota. Science 358:

Showing the most recent 10 out of 51 publications