Agency
National Institute of Health (NIH)
Institute
National Heart, Lung, and Blood Institute (NHLBI)
Type
Institutional National Research Service Award (T32)
Project #
2T32HL007599-11A1
Application #
2430581
Study Section
Special Emphasis Panel (ZHL1-CSR-Y (O1))
Project Start
1990-07-01
Project End
2003-06-30
Budget Start
1998-07-01
Budget End
1999-06-30
Support Year
11
Fiscal Year
1998
Total Cost
Indirect Cost
Name
Thomas Jefferson University
Department
Physiology
Type
Schools of Medicine
DUNS #
061197161
City
Philadelphia
State
PA
Country
United States
Zip Code
19107
Franke, Aaron S; Mooers, Susan U; Narayan, Srinivasa R et al. (2007) Myosin cross-bridge kinetics and the mechanism of catch. Biophys J 93:554-65
Stalker, Timothy J; Gong, Yulan; Scalia, Rosario (2005) The calcium-dependent protease calpain causes endothelial dysfunction in type 2 diabetes. Diabetes 54:1132-40
Scalia, Rosario; Stalker, Timothy J (2002) Microcirculation as a target for the anti-inflammatory properties of statins. Microcirculation 9:431-42
Booth, Gregory; Stalker, Timothy J; Lefer, Allan M et al. (2002) Mechanisms of amelioration of glucose-induced endothelial dysfunction following inhibition of protein kinase C in vivo. Diabetes 51:1556-64
Carey, L A; Perkowski, S Z; Lipsky, C L et al. (1997) Neutrophil recruitment as a factor limiting injury or promoting recovery from acute lung injury. Am J Physiol 272:H279-89
Carey, L A; Schuhl, R A; Gee, M H (1995) Microplate reader assay for measurement of opsonized zymosan-stimulated superoxide anion production by neutrophils. Biotechniques 19:824-9
Carey, L A; Albertine, K H; Gee, M H (1994) In vivo endotoxin exposure increases neutrophil counts without maintenance of primed superoxide anion release in short-term cultures of sheep bone marrow. J Leukoc Biol 56:145-50
Lefer, A M; Tsao, P S; Lefer, D J et al. (1991) Role of endothelial dysfunction in the pathogenesis of reperfusion injury after myocardial ischemia. FASEB J 5:2029-34
Tsao, P S; Lefer, A M (1990) Time course and mechanism of endothelial dysfunction in isolated ischemic- and hypoxic-perfused rat hearts. Am J Physiol 259:H1660-6