Chemotropic Function for NT-3 in CNS Development - Maryellen Daston

Abstract

Funding Agency

Agency: National Institute of Health (NIH)
Institute: National Institute of Neurological Disorders and Stroke (NINDS)
Type: Postdoctoral Individual National Research Service Award (F32)
Project #: 7F32NS009444-04
Application #: 2036603
Study Section: Neurology B Subcommittee 2 (NEUB)

Project Start: 1996-09-01
Project End
Budget Start: 1996-09-01
Budget End: 1997-03-06
Support Year: 4
Fiscal Year: 1996
Total Cost
Indirect Cost

Institution

Name: University of Cincinnati
Department: Physiology
Type: Schools of Medicine
DUNS #

City: Cincinnati
State: OH
Country: United States
Zip Code: 45221

Related projects


NIH 1996 F32 NS	Chemotropic Function for NT-3 in CNS Development Daston, Maryellen M. / University of Cincinnati
NIH 1996 F32 NS	Chemotropic Function for NT-3 in CNS Development Daston, Maryellen M. / Salk Institute for Biological Studies
NIH 1994 F32 NS	Mechanism of Chemotropic Induction of Axon Branching Daston, Maryellen M. / Salk Institute for Biological Studies
NIH 1993 F32 NS	Mechanism of Chemotropic Induction of Axon Branching Daston, Maryellen M. / Salk Institute for Biological Studies

Publications

Bastmeyer, M; Daston, M M; Possel, H et al. (1998) Collateral branch formation related to cellular structures in the axon tract during corticopontine target recognition. J Comp Neurol 392:1-18

Daston, M M; Bastmeyer, M; Rutishauser, U et al. (1996) Spatially restricted increase in polysialic acid enhances corticospinal axon branching related to target recognition and innervation. J Neurosci 16:5488-97

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