Agency
National Institute of Health (NIH)
Institute
National Institute on Aging (NIA)
Type
Research Project (R01)
Project #
5R01AG020684-15
Application #
6533965
Study Section
Immunobiology Study Section (IMB)
Program Officer
Fuldner, Rebecca A
Project Start
1987-04-01
Project End
2006-08-31
Budget Start
2002-09-01
Budget End
2003-08-31
Support Year
15
Fiscal Year
2002
Total Cost
$331,875
Indirect Cost
Name
University of California San Francisco
Department
Microbiology/Immun/Virology
Type
Schools of Medicine
DUNS #
073133571
City
San Francisco
State
CA
Country
United States
Zip Code
94143
Parsa, Jahan-Yar; Basit, Wajiha; Wang, Clifford L et al. (2007) AID mutates a non-immunoglobulin transgene independent of chromosomal position. Mol Immunol 44:567-75
Wang, Clifford L; Wang, Bruce B; Bartha, Gabor et al. (2006) Activation of an oncogenic microRNA cistron by provirus integration. Proc Natl Acad Sci U S A 103:18680-4
Wang, Clifford L; Yang, Desiree C; Wabl, Matthias (2006) Slow, stochastic transgene repression with properties of a timer. Genome Biol 7:R47
Wang, Clifford L; Wabl, Matthias (2005) Hypermutation rate normalized by chronological time. J Immunol 174:5650-4
Klasen, Maik; Spillmann, Freia J X; Marra, Giancarlo et al. (2005) Somatic hypermutation and mismatch repair in non-B cells. Eur J Immunol 35:2222-9
Glud, Sys Zoffmann; Sorensen, Annette Balle; Andrulis, Mindaugas et al. (2005) A tumor-suppressor function for NFATc3 in T-cell lymphomagenesis by murine leukemia virus. Blood 106:3546-52
Wang, Clifford L; Wabl, Matthias (2005) Mutational activity in cell line WEHI-231. Immunogenetics 56:849-53
Klasen, Maik; Spillmann, Freia J X; Lorens, James B et al. (2005) Retroviral vectors to monitor somatic hypermutation. J Immunol Methods 300:47-62
Wang, Clifford L; Wabl, Matthias (2004) DNA acrobats of the Ig class switch. J Immunol 172:5815-21
Wang, Clifford L; Hodgson, J Graeme; Malek, Tiffany et al. (2004) A murine leukemia virus with Cre-LoxP excisible coding sequences allowing superinfection, transgene delivery, and generation of host genomic deletions. Retrovirology 1:5

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