Agency
National Institute of Health (NIH)
Institute
National Cancer Institute (NCI)
Type
Research Project (R01)
Project #
5R01CA082858-05
Application #
6633501
Study Section
Metabolic Pathology Study Section (MEP)
Program Officer
Spalholz, Barbara A
Project Start
1999-07-01
Project End
2005-04-30
Budget Start
2003-05-01
Budget End
2005-04-30
Support Year
5
Fiscal Year
2003
Total Cost
$177,985
Indirect Cost
Name
Cleveland Clinic Lerner
Department
Type
DUNS #
017730458
City
Cleveland
State
OH
Country
United States
Zip Code
44195
Crosby, M E; Jacobberger, J; Gupta, D et al. (2007) E2F4 regulates a stable G2 arrest response to genotoxic stress in prostate carcinoma. Oncogene 26:1897-909
Mazumder, Suparna; Plesca, Dragos; Kinter, Michael et al. (2007) Interaction of a cyclin E fragment with Ku70 regulates Bax-mediated apoptosis. Mol Cell Biol 27:3511-20
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Crosby, Meredith E; Oancea, Marcela; Almasan, Alex (2004) p53 binding to target sites is dynamically regulated before and after ionizing radiation-mediated DNA damage. J Environ Pathol Toxicol Oncol 23:67-79
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Zheng, Yanhua; Shi, Yong; Tian, Changhai et al. (2004) Essential role of the voltage-dependent anion channel (VDAC) in mitochondrial permeability transition pore opening and cytochrome c release induced by arsenic trioxide. Oncogene 23:1239-47
Oancea, Marcela; Mani, Aruna; Hussein, Mohamad A et al. (2004) Apoptosis of multiple myeloma. Int J Hematol 80:224-31
DuPree, Erica L; Mazumder, Suparna; Almasan, Alexandru (2004) Genotoxic stress induces expression of E2F4, leading to its association with p130 in prostate carcinoma cells. Cancer Res 64:4390-3

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