Funding Agency

Agency
National Institute of Health (NIH)
Institute
National Institute of Diabetes and Digestive and Kidney Diseases (NIDDK)
Type
Research Project (R01)
Project #
3R01DK052371-01S1
Application #
2770625
Study Section
Cellular Biology and Physiology Subcommittee 1 (CBY)
Program Officer
Haft, Carol Renfrew
Project Start
1998-01-01
Project End
1998-03-31
Budget Start
1998-01-01
Budget End
1998-03-31
Support Year
1
Fiscal Year
1998
Total Cost
Indirect Cost

  Institution

Name
Harvard University
Department
Nutrition
Type
Schools of Public Health
DUNS #
082359691
City
Boston
State
MA
Country
United States
Zip Code
02115

  Publications

Simpson, Jeremy C; Griffiths, Gareth; Wessling-Resnick, Marianne et al. (2004) A role for the small GTPase Rab21 in the early endocytic pathway. J Cell Sci 117:6297-311
Akompong, T; Ramm, E; Chang, C et al. (1999) Immunological analysis of beta-thalassemic mouse intestinal proteins reveals up-regulation of sucrase-isomaltase in response to iron overload. J Nutr 129:949-52
Yu, J; Wessling-Resnick, M (1998) Structural and functional analysis of SFT, a stimulator of Fe Transport. J Biol Chem 273:21380-5
Yu, J; Wessling-Resnick, M (1998) Influence of copper depletion on iron uptake mediated by SFT, a stimulator of Fe transport. J Biol Chem 273:6909-15
Yu, J; Yu, Z K; Wessling-Resnick, M (1998) Expression of SFT (stimulator of Fe transport) is enhanced by iron chelation in HeLa cells and by hemochromatosis in liver. J Biol Chem 273:34675-8
Gutierrez, J A; Inman, R S; Akompong, T et al. (1998) Metabolic depletion inhibits the uptake of nontransferrin-bound iron by K562 cells. J Cell Physiol 177:585-92
Gutierrez, J A; Yu, J; Rivera, S et al. (1997) Functional expression cloning and characterization of SFT, a stimulator of Fe transport. J Cell Biol 139:895-905