IL-4 Inducible Protein in Lymphocyte Gene Expression

Boothby, Mark R.

Abstract

Funding Agency

• Agency: National Institute of Health (NIH)
• Institute: National Institute of General Medical Sciences (NIGMS)
• Type: First Independent Research Support & Transition (FIRST) Awards (R29)
• Project #: 5R29GM042550-04
• Application #: 3467716
• Study Section: Immunobiology Study Section (IMB)
• Project Start: 1989-07-01
• Project End: 1992-08-23
• Budget Start: 1992-07-01
• Budget End: 1992-08-23
• Support Year: 4
• Fiscal Year: 1992

Institution

• Name: Harvard University
• Type: Schools of Public Health
• DUNS #: 082359691
• City: Boston
• State: MA
• Country: United States
• Zip Code: 02115

Publications

Kovalik, J P; Ansari, B; Boothby, M (1996) Rapid commitment to negative selection of a thymocyte line independent of CD4 co-engagement. J Immunol 157:5290-9

Kim, J; Reeves, R; Rothman, P et al. (1995) The non-histone chromosomal protein HMG-I(Y) contributes to repression of the immunoglobulin heavy chain germ-line epsilon RNA promoter. Eur J Immunol 25:798-808

Whitley, M Z; Cheng, H L; Tomasi, T B et al. (1993) Distinct IL-4 response mechanisms of the MHC gene A alpha in different mouse B cell lines. Mol Immunol 30:821-32

Casey, L S; Lichtman, A H; Boothby, M (1992) IL-4 induces IL-2 receptor p75 beta-chain gene expression and IL-2-dependent proliferation in mouse T lymphocytes. J Immunol 148:3418-26