Agency
National Institute of Health (NIH)
Institute
National Institute of General Medical Sciences (NIGMS)
Type
Institutional National Research Service Award (T32)
Project #
5T32GM008291-04
Application #
3538413
Study Section
Special Emphasis Panel (SRC)
Project Start
1989-07-01
Project End
1994-06-30
Budget Start
1992-07-01
Budget End
1993-06-30
Support Year
4
Fiscal Year
1992
Total Cost
Indirect Cost
Name
Boston University
Department
Type
Schools of Arts and Sciences
DUNS #
604483045
City
Boston
State
MA
Country
United States
Zip Code
02118
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Dioum, Elhadji M; Rutter, Jared; Tuckerman, Jason R et al. (2002) NPAS2: a gas-responsive transcription factor. Science 298:2385-7
Rutter, J; Michnoff, C H; Harper, S M et al. (2001) PAS kinase: an evolutionarily conserved PAS domain-regulated serine/threonine kinase. Proc Natl Acad Sci U S A 98:8991-6
Rutter, J; Reick, M; Wu, L C et al. (2001) Regulation of clock and NPAS2 DNA binding by the redox state of NAD cofactors. Science 293:510-4
Brooks, C C; Tolan, D R (1994) A partially active mutant aldolase B from a patient with hereditary fructose intolerance. FASEB J 8:107-13
Morris, A J; Tolan, D R (1994) Lysine-146 of rabbit muscle aldolase is essential for cleavage and condensation of the C3-C4 bond of fructose 1,6-bis(phosphate). Biochemistry 33:12291-7
Brooks, C C; Tolan, D R (1993) Association of the widespread A149P hereditary fructose intolerance mutation with newly identified sequence polymorphisms in the aldolase B gene. Am J Hum Genet 52:835-40
Tolan, D R; Brooks, C C (1992) Molecular analysis of common aldolase B alleles for hereditary fructose intolerance in North Americans. Biochem Med Metab Biol 48:19-25
Brooks, C C; Buist, N; Tuerck, J et al. (1991) Identification of a splice-site mutation in the aldolase B gene from an individual with hereditary fructose intolerance. Am J Hum Genet 49:1075-81