The long-term goals and objectives of the Stanford University T35 program is to develop and enhance training opportunities for veterinary students interested in careers in biomedical, behavioral, and clinical research. The program will continue to provide intensive, short-term research training experiences for veterinary students from across the country that come to Stanford University for the summer. Each summer, eight first- through third-year veterinary students will be supported by the NIH and up to two more students will be supported by Stanford University. Students will spend 12 weeks at Stanford training in basic, behavioral, or clinical research aspects of the health-related sciences. Key elements of the research training plan include a mentored research project in a well funded and productive laboratory, research-related workshops, a journal club, research presentations, career development sessions with veterinarians who are at different levels of career development and that contribute to research in diverse ways, and research presentations at Stanford University and at the Merial-NIH National Veterinary Scholars Symposium. The program will encourage veterinary students to pursue research careers by exposure to and short-term involvement in the health-related sciences. Training will be of sufficient depth to enable students, upon completion of the program, to have a thorough exposure to the principles underlying the conduct of research. The program is designed to help veterinary students develop careers that will exert a sustained, powerful influence on biomedical, behavioral, and clinical research.
The United States needs more veterinary researchers. This program's primary objective is to help veterinary students develop research careers. The long-term effect of this program, and others like it, is strengthening of the nation's workforce in laboratory animal medicine, comparative pathology, and comparative medicine.
|Demars, Fanny; Clark, Kristen; Wyeth, Megan S et al. (2018) A single subconvulsant dose of domoic acid at mid-gestation does not cause temporal lobe epilepsy in mice. Neurotoxicology 66:128-137|
|Hofmann, Gabrielle; Balgooyen, Laura; Mattis, Joanna et al. (2016) Hilar somatostatin interneuron loss reduces dentate gyrus inhibition in a mouse model of temporal lobe epilepsy. Epilepsia 57:977-83|
|Zhang, Wei; Thamattoor, Ajoy K; LeRoy, Christopher et al. (2015) Surviving mossy cells enlarge and receive more excitatory synaptic input in a mouse model of temporal lobe epilepsy. Hippocampus 25:594-604|
|Scharfman, Helen E; Buckmaster, Paul S (2014) Preface. Adv Exp Med Biol 813:xv-xviii|
|Toyoda, Izumi; Bower, Mark R; Leyva, Fernando et al. (2013) Early activation of ventral hippocampus and subiculum during spontaneous seizures in a rat model of temporal lobe epilepsy. J Neurosci 33:11100-15|
|Heng, Kathleen; Haney, Megan M; Buckmaster, Paul S (2013) High-dose rapamycin blocks mossy fiber sprouting but not seizures in a mouse model of temporal lobe epilepsy. Epilepsia 54:1535-41|
|Ma, Yunyong; Ramachandran, Anu; Ford, Naomi et al. (2013) Remodeling of dendrites and spines in the C1q knockout model of genetic epilepsy. Epilepsia 54:1232-9|
|Buckmaster, Paul S; Haney, Megan M (2012) Factors affecting outcomes of pilocarpine treatment in a mouse model of temporal lobe epilepsy. Epilepsy Res 102:153-9|
|Lew, Felicia H; Buckmaster, Paul S (2011) Is there a critical period for mossy fiber sprouting in a mouse model of temporal lobe epilepsy? Epilepsia 52:2326-32|
|Buckmaster, Paul S; Lew, Felicia H (2011) Rapamycin suppresses mossy fiber sprouting but not seizure frequency in a mouse model of temporal lobe epilepsy. J Neurosci 31:2337-47|
Showing the most recent 10 out of 13 publications