Agency
National Institute of Health (NIH)
Institute
National Cancer Institute (NCI)
Type
Research Project (R01)
Project #
5R01CA040847-10
Application #
2090329
Study Section
Metabolic Pathology Study Section (MEP)
Project Start
1985-02-01
Project End
1995-06-30
Budget Start
1995-02-01
Budget End
1995-06-30
Support Year
10
Fiscal Year
1995
Total Cost
Indirect Cost
Name
University of Nebraska Medical Center
Department
Internal Medicine/Medicine
Type
Schools of Medicine
DUNS #
City
Omaha
State
NE
Country
United States
Zip Code
68198
Liu, M; Pelling, J C; Ju, J et al. (1998) Antioxidant action via p53-mediated apoptosis. Cancer Res 58:1723-9
Dhanwada, K R; Dickens, M; Neades, R et al. (1995) Differential effects of UV-B and UV-C components of solar radiation on MAP kinase signal transduction pathways in epidermal keratinocytes. Oncogene 11:1947-53
Betz, N A; Pelling, J C (1995) Ha-ras p21-GTP levels remain constant during primary keratinocyte differentiation. Mol Carcinog 12:66-76
Liu, M; Pelling, J C (1995) UV-B/A irradiation of mouse keratinocytes results in p53-mediated WAF1/CIP1 expression. Oncogene 10:1955-60
Chakravarti, D; Pelling, J C; Cavalieri, E L et al. (1995) Relating aromatic hydrocarbon-induced DNA adducts and c-H-ras mutations in mouse skin papillomas: the role of apurinic sites. Proc Natl Acad Sci U S A 92:10422-6
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Fujii, H; Egami, H; Chaney, W et al. (1990) Pancreatic ductal adenocarcinomas induced in Syrian hamsters by N-nitrosobis(2-oxopropyl)amine contain a c-Ki-ras oncogene with a point-mutated codon 12. Mol Carcinog 3:296-301

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