Agency
National Institute of Health (NIH)
Institute
Eunice Kennedy Shriver National Institute of Child Health & Human Development (NICHD)
Type
Research Project (R01)
Project #
2R01HD026594-06A2
Application #
2199996
Study Section
Special Emphasis Panel (ZRG2-HED-2 (01))
Project Start
1996-03-01
Project End
2000-02-29
Budget Start
1996-03-01
Budget End
1997-02-28
Support Year
6
Fiscal Year
1996
Total Cost
Indirect Cost
Name
Dana-Farber Cancer Institute
Department
Type
DUNS #
149617367
City
Boston
State
MA
Country
United States
Zip Code
02215
Gross, Vera S; Hess, Mailee; Cooper, Geoffrey M (2005) Mouse embryonic stem cells and preimplantation embryos require signaling through the phosphatidylinositol 3-kinase pathway to suppress apoptosis. Mol Reprod Dev 70:324-32
Zilz, Alexandra; Cooper, Geoffrey M (2004) A binding site for germ cell nuclear factor within c-mos regulatory sequences. Mol Reprod Dev 67:55-64
Gross, Vera S; Cooper, Geoffrey M (2002) Functional analysis of sperm from c-mos(-/-) mice. Mol Reprod Dev 62:519-24
Lin, H B; Jurk, M; Gulick, T et al. (1999) Identification of COUP-TF as a transcriptional repressor of the c-mos proto-oncogene. J Biol Chem 274:36796-800
Xu, W; Cooper, G M (1995) Identification of a candidate c-mos repressor that restricts transcription of germ cell-specific genes. Mol Cell Biol 15:5369-75
Pal, S K; Torry, D; Serta, R et al. (1994) Expression and potential function of the c-mos proto-oncogene in human eggs. Fertil Steril 61:496-503
Gebauer, F; Xu, W; Cooper, G M et al. (1994) Translational control by cytoplasmic polyadenylation of c-mos mRNA is necessary for oocyte maturation in the mouse. EMBO J 13:5712-20
Yamauchi, N; Kiessling, A A; Cooper, G M (1994) The Ras/Raf signaling pathway is required for progression of mouse embryos through the two-cell stage. Mol Cell Biol 14:6655-62
Pal, S K; Crowell, R; Kiessling, A A et al. (1993) Expression of proto-oncogenes in mouse eggs and preimplantation embryos. Mol Reprod Dev 35:8-15
Zinkel, S S; Pal, S K; Szeberenyi, J et al. (1992) Identification of a negative regulatory element that inhibits c-mos transcription in somatic cells. Mol Cell Biol 12:2029-36

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